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Title : New Study Confirms Wheat-Autism Link
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New Study Confirms Wheat-Autism Link

As the autism epidemic continues to accelerate, one delos lesser-known factors that contribute mostly goes unnoticed: the trigoconsumo.

A new study published in the open access journal PLoS esrevitalizar the debate on what the main causes of autism epidemic laacelerar. While many in the medical convencionalestablecimiento, failing to identify a single cause, apathy labeled with "idiopathic", ie, cause unknown, opeor condition "caused by defective genes unknown," there is a growing awareness greater than a multitude of environmental factors including vaccines chemical cesareans , antibiotics, genetically modified foods, and sonesencial food intolerances in the understanding and treatment of this disturbing condition.
The point, therefore, is not to prove one thing the cause (eg "autism genes"), and another not the cause (eg vaccines), as if fueraestrictamente some kind of sport or academic more beyond time, rather areconocer possible contributing factors, and remove siempreposible as a precaution.

The new study entitled, " markers of celiac disease and gluten sensitivity in children with autism ," explored the possible association between sensitivity yautismo gluten, which previous research has confirmed only inconsistently.


described his goal as follows: "[T] or evaluate the inmunológicaal reactivity gluten in patients pediatric diagnosed with strict segúncriterios autism and assess the possible link between autism andthe celiac disease. "
they found that children with autism had significantly higher levels of IgG antibodies against gliadin, lainmunotóxicos primary protein in wheat, compared to controls saludables.También found that the IgG anti-gliadin response was significantly higher in autistic children withthe symptoms gastrointestinal compared to those without. losLos elevated IgG antibodies indicate: 1) that your immune system estala identification of gliadin proteins as "other". 2) estardebidamente gliadin is not broken down by digestive processes and are entrandoa intestinal tract through a compromised ( "leaky gut") in the blood.
It has been theorized that when the immune system is anticuerposcontra gliadin, these antibodies cross with self-estructurasdentro nervous system reaction. Known as molecular mimicry, this descomponerdel immunological self-tolerance can help wide range of neurological problems, including neuropathy, ataxia, seizures and changes in neurobehavioral, including mania , schizophrenia and autism. The anti-gliadin antibodies are therefore a possible cause of autoimmune neurological damage. A study published in Journal of Immunology in 2007 found that anti-gliadin antibodies cross-react with protein neurological class known as synapsin I, providing a molecular mechanism behind how immune unareacción to gliadin could help neurológicadegeneración.

Beyond Celiac Disease: Wheat causes much more harm than commonly understood

When the researchers analyzed levels of certain celíacamarcadores disease blood (ie, deamidated gliadin antibodies and TG2) which hicieronno differ between patients and controls. Moreover, not huboasociación observed between genetic locus HLA-DQ2 celiac disease susceptibility and antibodies anti-gliadin increased. Unhallazgo This is important because many look for wheat intolerance or sensitivity as a rare disorder without an action mechanism well established celiac comoenfermedad. They prefer not to believe that wheat, which is unagrano glorified in richer countries, could contribute to a wide range of diseases. Our project, for example, has identified more than 200potencial adverse health effects of gluten consumption. One does not have to be, therefore, the definition celiac clásicaenfermedad, or "wheat allergy," experiencing adversosasociado with eating gluten grain effects.

The authors of the study concluded:

A subgroup of children with autism shows increased inmunereactividad gluten, the mechanism of which appears to be different from that in celiac disease. The response of anti-gliadin antibodies aumentoy its association with GI symptoms points to a potencialprovocada by immunological abnormalities permeability and / or intestinal mechanism inthe affected children.

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